A patient diagnosed with an ankle sprain is prescribed NSAIDs. What is the primary expected pharmacological action of NSAIDs in this situation?

A. Enhancement of gastrointestinal motility and digestion: NSAIDs do not improve digestion or gastrointestinal motility. In fact, they can irritate the gastric mucosa and increase the risk of ulcers or GI bleeding, making this unrelated to their therapeutic purpose for an ankle sprain. B. Inhibition of inflammatory processes and reduction of pain associated with soft tissue injury: NSAIDs block cyclooxygenase (COX) enzymes, reducing the production of prostaglandins that mediate inflammation, pain, and swelling. This mechanism directly addresses the discomfort and tissue inflammation associated with an ankle sprain. C. Promotion of blood clotting and platelet aggregation: NSAIDs, particularly nonselective ones like ibuprofen, can inhibit platelet aggregation and increase bleeding risk. Promoting clotting is not their pharmacologic effect and is contrary to their known actions. D. Stimulation of immune response and antibody production: NSAIDs do not stimulate the immune system or antibody production. Their primary action is anti-inflammatory and analgesic, rather than immunomodulatory.