Analyze the following clinical manifestations and determine whether they are characteristic of Anticholinergic or Cholinergic Toxicity.

Hypertension: Seen in anticholinergic toxicity due to decreased parasympathetic tone and possible unopposed sympathetic stimulation. The body's inability to relax vascular tone may contribute to elevated blood pressure. Urinary Retention: A classic feature of anticholinergic toxicity. Inhibition of muscarinic receptors leads to impaired detrusor muscle contraction, making urination difficult or impossible. Hyperthermia: Results from inhibited sweating (anhidrosis), which is a hallmark of anticholinergic toxicity. Without the ability to cool through evaporation, body temperature rises dangerously. Diaphoresis: Excessive sweating is mediated by muscarinic receptor activation in cholinergic toxicity. It's often part of the SLUDGE symptoms seen in organophosphate poisoning or cholinesterase inhibitor overdose. Bronchoconstriction: Caused by overstimulation of muscarinic receptors in the airways, leading to narrowed bronchi and increased respiratory secretions — a dangerous feature of cholinergic excess. Salivation: Prominent in cholinergic toxicity due to unopposed parasympathetic activation. It may occur along with lacrimation, bradycardia, and other signs of excessive cholinergic stimulation.