Exhibits The nurse determines that the client is experiencing toxicity and notifies the HCP. Which action(s) should the nurse anticipate performing to manage digoxin toxicity? Select all that apply.

A. Monitor radial pulse: Monitoring the radial pulse is critical because digoxin toxicity can cause significant bradycardia and arrhythmias. Frequent pulse checks help assess cardiac status and detect worsening toxicity. B. Administer furosemide PO: Furosemide is a loop diuretic that can worsen hypokalemia, which increases the risk of digoxin toxicity. Administering it without correcting potassium levels can exacerbate the problem, so this is not an appropriate immediate action. C. Give IV fluids: While fluids may be necessary for some conditions, IV fluids do not specifically treat digoxin toxicity. In cases of toxicity, careful fluid management is needed, especially in heart failure patients, to avoid fluid overload. D. Stop digoxin: Discontinuing digoxin is essential to prevent further accumulation and worsening toxicity, especially when signs and symptoms such as bradycardia, nausea, and visual changes are present. E. Administer digoxin immune fab: Digoxin immune fab is the antidote for severe digoxin toxicity. It binds free digoxin, neutralizing its effects, and is indicated in life-threatening toxicity or symptomatic bradyarrhythmias. F. Give atropine: Atropine is used to treat symptomatic bradycardia caused by digoxin toxicity by blocking parasympathetic stimulation and increasing heart rate. G. Administer potassium IV: The client's hypokalemia is a significant contributing factor to the toxicity. Administering potassium, likely via IV to replenish the low levels quickly, will help reverse the toxicity and protect the heart. H. Monitor potassium levels: Close monitoring of potassium is vital because hypokalemia increases digoxin toxicity risk, and potassium levels may fluctuate with treatment, affecting cardiac function.