Patient Data. Exhibits Click to mark whether the assessment finding represents a therapeutic result of the sodium bicarbonate administered a non-therapeutic side-effect, or an unrelated finding. Each row must have one option selected.

Rationale: Blood pressure 113/72 mm Hg: Fluid resuscitation and correction of acidosis improved vascular tone and cardiac output, restoring perfusion. This normalization of blood pressure reflects effective volume replacement and stabilization after severe dehydration. Bicarbonate 22 mEq/L: Sodium bicarbonate directly replenishes bicarbonate ions, correcting metabolic acidosis. The return of HCO₃⁻ to the normal reference range demonstrates a successful pharmacologic effect. pH 7.35: Initial pH was severely acidotic at 7.22. Following sodium bicarbonate, normalization to 7.35 shows resolution of metabolic acidosis. This confirms adequate buffering and compensation, representing the intended therapeutic outcome. Capillary refill 2 seconds: Improved perfusion results from fluid therapy and correction of acidosis, which enhances vascular responsiveness and tissue oxygen delivery. The change from 5 seconds to 2 seconds indicates recovery of adequate circulation. Heart rate 77 beats/minute: The initial tachycardia of 102 bpm reflected hypovolemia and acidosis. With restoration of intravascular volume and acid–base balance, heart rate decreased to a normal level, showing stabilization of cardiovascular function. Sodium 152 mEq/L: Hypernatremia developed as a nontherapeutic effect. Sodium bicarbonate contains a high sodium load, which, combined with IV fluids, can raise serum sodium. This increases risk for neurologic complications such as confusion or seizures if not monitored. 400 mL urine output: Improved urine output reflects renal perfusion recovery due to IV fluid resuscitation, not sodium bicarbonate. The restoration of intravascular volume from isotonic fluids is the primary factor here, making this an unrelated finding.