Patient Data The nurse reviews the client's data. Drag one client finding and one rationale to complete the sentence. The client is at risk for developing _____ due to a(n) _____ .

• Alcohol withdrawal: The client’s history of chronic alcohol use for 10 years and her current presentation—tremors, diaphoresis, nausea, agitation, disorientation, and elevated vital signs—are classic signs of alcohol withdrawal. These symptoms typically begin within hours of reduced alcohol intake and can progress to severe complications if untreated. • Psychomotor agitation: While the client is exhibiting psychomotor agitation (hand tremors, restlessness, fidgeting), this is a symptom of alcohol withdrawal rather than the underlying condition itself. Psychomotor agitation alone does not explain the cause of her altered mental status or autonomic changes. • Hallucinations: Hallucinations can occur in severe alcohol withdrawal (delirium tremens), but the client has not yet demonstrated perceptual disturbances such as visual or auditory hallucinations. Her current presentation is primarily characterized by autonomic hyperactivity and tremors, making hallucinations a possible future complication rather than an immediate diagnosis. • Neurotransmitter imbalance: Chronic alcohol use disrupts the balance of inhibitory (GABA) and excitatory (glutamate) neurotransmitters. Abrupt cessation leads to a relative overactivity of excitatory pathways, producing tremors, agitation, tachycardia, and hypertension. This imbalance explains her psychomotor agitation, confusion, and autonomic instability. • Increase in acetaldehyde levels: Acetaldehyde accumulation occurs during alcohol metabolism and contributes to hangover symptoms, but it is not the main mechanism driving withdrawal. Her acute symptoms are related to central nervous system hyperexcitability from neurotransmitter imbalance, not residual acetaldehyde toxicity. • Withdrawal-related dopamine surge: While dopamine may fluctuate during withdrawal, the predominant pathophysiology involves excessive excitatory neurotransmission due to GABA/glutamate imbalance. Focusing solely on dopamine changes does not fully account for her autonomic hyperactivity, tremors, and disorientation.