The nurse is caring for a client in the emergency department. Exhibits For each client finding, click to specify if the finding is consistent with pyelonephritis or glomerulonephritis. Each finding may support more than one disease process or none at all. There must be at least 1 selection in every column. There does not need to be a selection in every row.

Rationale: Blood pressure: In glomerulonephritis, blood pressure elevation is commonly seen due to fluid retention and impaired kidney function, which leads to increased vascular resistance. This is more pronounced than in pyelonephritis, where blood pressure changes may be less severe. Respiratory rate: Both pyelonephritis and glomerulonephritis can cause an elevated respiratory rate. In pyelonephritis, it may be due to systemic infection or respiratory distress caused by fluid accumulation or sepsis. In glomerulonephritis, respiratory rate can increase due to fluid retention, leading to pulmonary congestion or difficulty breathing. Urinary output amount: Both conditions can cause decreased urinary output. In pyelonephritis, this may occur due to kidney inflammation or infection affecting kidney function. In glomerulonephritis, decreased output is due to glomerular damage that impairs filtration, leading to oliguria or anuria. Edema: Edema, particularly periorbital and in the extremities, is a hallmark of glomerulonephritis. The glomerular filtration barrier is damaged, causing protein loss in the urine and subsequent fluid retention. In contrast, edema is less common in pyelonephritis and usually occurs only in severe or advanced cases. Pain location: Flank pain is a key feature of pyelonephritis due to kidney infection or inflammation. The pain is usually localized to the back or side, in the area where the kidneys are located. Glomerulonephritis typically does not cause significant flank pain; it is more associated with systemic symptoms like edema, hematuria, and hypertension.