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    W126 n241 med surg proctored Exam Swedish insistute

    The nurse notices the rhythm below on the heart monitor. When the client is assessed, the client is found to be unresponsive with no pulse. What should the nurse's next action?

    Explanation & Rationale

    Ventricular tachycardia is a lethal cardiac arrhythmia characterized by wide, bizarre QRS complexes and the absence of P-waves. When this rhythm occurs without a palpable pulse, it indicates a total loss of cardiac output and effective mechanical function, necessitating immediate resuscitative efforts to restore perfusion and prevent irreversible biological death. A. Pulseless ventricular tachycardia is a "shockable" cardiac arrest rhythm. The Advanced Cardiovascular Life Support (ACLS) protocol dictates the immediate initiation of high-quality chest compressions (CPR) and rapid defibrillation. Defibrillation delivers an unsynchronized shock to depolarize the entire myocardium simultaneously, allowing the heart's natural pacemaker to resume a viable rhythm. B. Ignoring this rhythm is a critical failure of care, as ventricular tachycardia is never benign, especially when the patient is unresponsive and pulseless. This rhythm represents a state of clinical death. Without immediate intervention, the patient will suffer permanent brain damage within minutes and progress rapidly to asystole and biological death. C. Electrical cardioversion is a synchronized shock delivered on the R-wave of the QRS complex, used for patients who have a pulse but are hemodynamically unstable. In a pulseless patient, the machine cannot track an R-wave reliably, and a synchronized shock is inappropriate. Pulseless patients require unsynchronized defibrillation, not synchronized cardioversion. D. Intravenous verapamil is a calcium channel blocker used for certain supraventricular tachycardias in stable patients. Administering it to a patient in ventricular tachycardia, particularly one without a pulse, is dangerous and contraindicated. It would cause profound vasodilation and further decrease myocardial contractility, worsening the patient's already terminal hemodynamic state.

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