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    Ati pn adult medical surgical 2023 proctored exam
    Select All That Apply

    Which of the following findings require immediate follow-up by the nurse? (Select all that apply.)

    Explanation & Rationale

    Diabetic ketoacidosis involves profound metabolic disturbances including dehydration, electrolyte depletion, and hyperglycemia. While all clinical signs reflect the severity of the disease, life-threatening complications like critical electrolyte imbalances and rapid, massive fluid loss require immediate intervention to stabilize cardiac rhythm and restore hemodynamic balance. Choice A rationale: Drowsiness is a expected neurological manifestation of severe metabolic acidosis and dehydration in DKA. While concerning, it is secondary to the metabolic crisis, and the nurse must prioritize fixing the underlying electrolyte and fluid status to improve the patient's level of consciousness. Choice B rationale: Abdominal pain is a common symptom in patients with DKA, often caused by gastric stasis or metabolic acidosis. It requires monitoring and pain management, but it is not as acutely life-threatening as the critical electrolyte levels identified in the laboratory results. Choice C rationale: Polydipsia, or extreme thirst, is a clinical consequence of osmotic diuresis caused by severe hyperglycemia. While indicative of significant intracellular dehydration, it is a symptomatic marker rather than an acute medical emergency that surpasses the need for electrolyte replacement. Choice D rationale: Fruity breath is a classic, non-urgent finding in DKA caused by the presence of acetone. This diagnostic indicator confirms the presence of ketosis but does not require immediate, independent nursing follow-up beyond the standard management plan for the patient's ketoacidosis. Choice E rationale: Dry mucous membranes and poor skin turgor are physical manifestations of severe dehydration. These findings provide evidence for the clinical severity of the condition but are treated through the ongoing, prescribed fluid resuscitation protocol rather than requiring a separate, immediate nurse-led intervention. Choice F rationale: A urine output of 500 mL in 30 minutes indicates extreme polyuria, likely due to osmotic diuresis or potentially catheter drainage of a previously full bladder. This rapid volume loss necessitates immediate assessment of fluid status to prevent hypovolemic shock. Choice G rationale: A blood glucose of 700 mg/dL is severely elevated. While this is the primary driver of DKA, the standard treatment is a continuous insulin infusion. This is a controlled, protocol-driven intervention rather than an acute, immediate-follow-up crisis requiring separate nursing priority. Choice H rationale: A potassium level of 2.9 mEq/L is critically low. Hypokalemia in DKA is life-threatening as it predisposes the patient to fatal cardiac arrhythmias. Potassium must be replaced immediately before or concurrently with insulin, making this a top nursing priority. Choice I rationale: A sodium level of 131 mEq/L indicates mild hyponatremia, common in DKA due to osmotic shifts. While it requires correction during rehydration, it is not as acutely dangerous as the critical potassium and magnesium imbalances present in this specific patient case. Choice J rationale: A magnesium level of 1.0 mEq/L is critically low. Severe hypomagnesemia can trigger cardiac arrhythmias and seizures, and it interferes with potassium correction. Immediate replacement is essential for patient safety, ranking it as a priority finding requiring urgent follow-up. Choice K rationale: An anion gap of 22 mEq/L indicates significant metabolic acidosis. This is the hallmark diagnostic finding for DKA and is addressed through the standard insulin and fluid protocol. It does not require a separate, immediate follow-up action by the nurse. Choice L rationale: Positive urine ketones confirm the diagnosis of DKA. This is a foundational diagnostic finding consistent with the patient's condition, but it is an expected indicator that is managed through the overarching treatment plan for the ketoacidotic crisis.

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