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    Med Surg Proctored Exam (shock) Samuel Merit University)

    You suspect your patient who is in neurogenic shock to have which of the following findings?

    Explanation & Rationale

    Choice A rationale Tachycardia is typically a compensatory mechanism in most types of shock, such as hypovolemic or septic shock, where the body increases the heart rate to maintain cardiac output. However, in neurogenic shock, the loss of sympathetic nervous system tone prevents this response. A heart rate of 115 beats per minute would suggest a different etiology or a compensatory phase that is physiologically absent when the cervical or high thoracic spinal cord is injured. Choice B rationale Neurogenic shock is characterized by bradycardia because the loss of sympathetic tone allows the parasympathetic nervous system to dominate, slowing the heart rate significantly. A heart rate of 46 beats per minute is a classic finding in this condition. The normal heart rate range is 60 to 100 beats per minute. Without sympathetic signals from the brain, the heart cannot increase its rate to compensate for the massive vasodilation occurring in the systemic vasculature. Choice C rationale Hypertension is not a finding associated with neurogenic shock; instead, patients typically present with profound hypotension. The loss of sympathetic tone causes massive vasodilation, which results in a significant drop in systemic vascular resistance and blood pressure. A blood pressure of 150/85 is considered elevated or at the upper limit of normal, whereas neurogenic shock patients require vasopressors and fluids to keep the systolic pressure above 90 mmHg. Choice D rationale While respiratory status can be affected if the spinal injury is high, a carbon dioxide level of 50 mmHg is not a specific diagnostic finding for neurogenic shock itself. Normal carbon dioxide levels range from 35 to 45 mmHg. While hypoventilation might occur if the diaphragm is compromised, the primary hemodynamic markers of neurogenic shock are the combination of hypotension and bradycardia. Carbon dioxide elevation is a secondary respiratory complication rather than a primary circulatory indicator.

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